2 edition of utilization by rats of the energy in riboflavin deficient diets found in the catalog.
utilization by rats of the energy in riboflavin deficient diets
in Urbana, Ill
Written in English
|Statement||by Joseph Bernsohn ...|
|LC Classifications||QP801.V5 B54|
|The Physical Object|
|Pagination||7,  p.|
|LC Control Number||a 44002049|
Summary. It has been demonstrated that a severe riboflavin deficiency in the rat results in a partial paralysis of the legs. This type of paralysis is produced more easily on a high fat ration and it is prevented by the daily feeding of crystalline by: Methods: Weanling male rats were randomly assigned either to control, riboflavin-replete (RR) or to experimental, riboflavin-deficient (RD) versions of the AING diet for 13 d, at which time a constant infusion of [13 C]-formate was carried out to ascertain the effects of deficiency Cited by: 4.
2. McCabe H. Riboflavin Deficiency in Cystic Fibrosis: Three Case Reports. J Hum Nutr Diet. Oct;14(5) Three cases of clinical riboflavin deficiency are reported in children aged years attending a regional Cystic Fibrosis clinic. Riboflavin deficiency presented as angular stomatitis in . A study was conducted to assess the effects of riboflavin deficiency and riboflavin supplementation on carcinogen-DNA binding. After 12 wk on a riboflavin-sufficient or a riboflavin-deficient diet male Wistar rats were administered 3 H-labelled benzo[a]pyrene (BP) ip. [ 3 H]BP was given either at a uniform dose of μCi/rat irrespective of body weight or at a dose adjusted to body weight.
Experiment 3: Analysis of Riboflavin in Energy Drinks INTRODUCTION Learning Goals Riboflavin is a common component in many different brands of energy drinks. Riboflavin, also known as vitamin B 2, is a water-soluble B vitamin and an integral component of . Iron absorption and daily loss of Fe were measured in riboflavin-deficient (B2-) Norwegian hooded rats and controls (B2+). Animals were fed on a test meal extrinsically labelled with 59Fe and whole-body radioactivity measured for 15 d. Riboflavin deficiency led to a reduction in the percentage of the 59Fe dose absorbed and an increased rate of 59Fe loss.
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The increase in activation coefficient (stimulated: basal activity) of erythrocyte NAD(P)H 2:glutathione oxidoreductase (EC ) and reduction in hepatic flavin concentration which occurred in riboflavin-deficient weanling rats were not markedly or consistently affected by differences in the concentration of lipid in the diet nor by differences in the total proportion of saturated or Cited by: 1.
Author(s): Bernsohn,Joseph, Title(s): The utilization by rats of the energy in riboflavin deficient diets,by Joseph Bernsohn Country of Publication: United States Publisher: Urbana, Ill., A control group of 20 rats, 10 belonging to a strain with a high efficiency of food utilization, and 10 to one with a low efficiency, received a diet of purified cas sucr salt mixture 4, lard 8 and cod liver oil 2, with adequate daily supplements of thiamine (vitamin B1), riboflavin, vitamin B6, Ca pantothenate and by: 2.
Lipid metabolism in riboflavin-deficient rats. utilization of these fatty acids in their tissues providing approximately and of the total energy of the diet.
Weanling rats received purified diets containing 10 per cent. lard supplemented with fat-soluble and B vitamins. Separate groups received normal lard, or rancid lard aerated at 90° C. for from 30 to 50 or for hr., and sometimes the food intake of those receiving non-rancid lard was restricted to that of those receiving rancid by: 8.
deficient diet, and the control group was fed the same diet supplemented with 6 mg riboflavin per kilogram. The feeding of the control group was restricted so that.
This study investigated the effects of riboflavin on energy metabolism in hypoxic mice. Kunming mice were fed diets containing riboflavin at doses of 6, 12, 24 and 48 mg/kg, respectively for 2 weeks before exposure to a simulated altitude of m for 8 h.
Changes of riboflavin status and energy metabolism were assessed biochemically. Riboflavin (B2) plays a central role in intermediary metabolism and energy release; little is known about riboflavin requirements or pathophysiological consequences of deficiency states in Author: Lucas A, Powers Hj, Duerden J, Bates Cj.
Riboflavin deficiency is widespread in many regions over the world, especially in underdeveloped countries. In this study, we investigated the effects of riboflavin deficiency on protein Cited by: 9. There was a tendency of higher whole body riboflavin concentration in fish fed high‐lipid diets.
Based on growth, absence of deficiency signs and maximal tissue saturation of riboflavin, it can be concluded that the requirement for riboflavin was met by the natural riboflavin content in the raw materials of the by: Nitrogen balances and tissue nitrogen levels of riboflavin deficient and supplemented male Sprague Dawley Crl:Cd rats were compared during successive Author: Pirkko R.
Turkki, Jane Buratto, Mark Corteville, Mary Patricia Driscoll. Abstract. Riboflavin deficiency has been studied in many animal species and always results in failure to grow. This fact might be anticipated in view of the importance of.
Riboflavin deficiency will not occur if the diet contains meat or dairy products (Watson, ). While it is unlikely that a clinical riboflavin deficiency would be seen in cats today, it is not impossible for it to occur in dogs. Most commercial pet foods are supplemented with synthetic riboflavin (Hand et al., ).
A diet containing at least kcal ME/g (metabolizable energy/gram) ( kJ ME/g) will meet the energy requirement for maintenance and growth if rats are allowed free access to food and the diet is not deficient in other nutrients.
Five percent fat is the minimum requirement for all stages of life. Lactococcus lactis is a commonly used starter strain that can be converted from a vitamin B 2 consumer into a vitamin B 2 ‘factory’ by over-expressing its riboflavin biosynthesis genes.
The present study was conducted to assess in a rat bioassay the response of riboflavin produced by GM or native lactic acid bacteria (LAB).Cited by: The riboflavin in most foods is in the form of FAD, although the main form in eggs and milk is free riboflavin. About 95% of riboflavin in the form of FAD or FMN from food is bioavailable up to a maximum of about 27 mg of riboflavin per meal or dose [ 3 ].The bioavailability of free riboflavin is similar to that of FAD and FMN [ 9, 10 ].
Weanling male rats were fed a riboflavin deficient diet for 5 weeks and enlargement of the liver mitochondria and discontinuity of the outer membrane were observed.
The content of phospholipids was slightly increased in the deficient mitochondria, and decrease in phosphatidylcholine and increase in phosphatidylethanolamine were shown by: Leclerc (b) fed diets containing l, 2, 4 or 6 mg riboflavin/kg to gestating rats and reported larger litter sizes at levels greater than l mg/kg.
No effects on prog- eny whole body riboflavin pool or on dam BW, liver riboflavin concentration, or weight gain were noted. D.B. McCormick, in Encyclopedia of Food Sciences and Nutrition (Second Edition), Deficiency Causes and Symptoms. Pure, uncomplicated riboflavin deficiency is probably never encountered in patients, but is accompanied by multiple nutrient deficiencies.
Ariboflavinosis can result from such primary and secondary factors as commonly affect supply or utilization of other nutrients as well. fed the riboflavin-deficient diet supplemented with riboflavin antagonist, galactoflavin, to a level of 60 mg/kg.
In another series, a group of animals was starved for 2 days and then killed. In a final series, a group of rats was fed a riboflavin-deficient diet supplemented. Riboflavin, also known as vitamin B 2, is a vitamin found in food and used as a dietary supplement.
Food sources include eggs, green vegetables, milk and other dairy product, meat, mushrooms, and almonds. Some countries require its addition to grains. As a supplement it is used to prevent and treat riboflavin deficiency and prevent migraines.
It may be given by mouth or injection.1–3: In rats, if mothers are fed on a riboflavin deficient diet, many of their progeny are affected with con- genital malformations, including defective deve-lopment of the mandible and cleft palate (Warkany and Schraffenberger, ; Bauer, ).
Nicotinic Acid:In nicotinic acid or niacin deficiency the tongue becomes markedly painful, beefy red.Abstract. The clinical consequences of riboflavin deficiency in humans were first described by Sebrell and Butler ().
On a diet which furnished only mg of riboflavin daily, 10 of 18 adult women developed oral lesions that disappeared following riboflavin by: 3.